#270 ‒ Journal club with Andrew Huberman: metformin as a geroprotective drug, the power of belief, and how to read scientific papers

Sep 11, 2023 Episode Page ↗
Overview

Peter Attia, MD, and Andrew Huberman, Professor of Neurobiology at Stanford, host a journal club discussing a metformin study and how beliefs influence drug effects. They also share their methodologies for interpreting scientific papers.

At a Glance
29 Insights
2h 16m Duration
14 Topics
9 Concepts

Deep Dive Analysis

Introduction to Journal Club and Paper Interpretation

Metformin: History, Mechanism, and Use in Type 2 Diabetes

Defining Insulin Resistance and its Causes

Metformin's Geroprotective Potential: The 2014 Bannister Study

Critique of Bannister Study: The Role of Informative Censoring

The 2022 Keys et al. Study Reassessing Metformin's Survival Advantage

Interpreting Epidemiological Data: Crude Mortality and Hazard Ratios

Limitations of Epidemiology and Personal Experience with Metformin

Current Thoughts on Metformin for Longevity and Caloric Restriction

Peter and Andrew's Process for Reading Scientific Papers

Understanding Belief Effects vs. Placebo Effects

Neurobiology of Nicotine and its Cognitive Enhancement

The Perl et al. Study: Belief's Impact on Nicotine's Brain Effects

Implications of Belief-Dependent Drug Responses in Medicine

Metformin Mechanism

Metformin inhibits complex one of the mitochondria, which changes the ratio of adenosine monophosphate to adenosine diphosphate. This action reduces the amount of glucose the liver puts out, helping to lower blood glucose levels in diabetics.

Insulin Resistance

This occurs when cells, primarily muscle cells, become less responsive to insulin, requiring more insulin to transport glucose into the cell. It is often caused by intramuscular fat accumulation, inactivity, sleep deprivation, and hypercortisolemia.

Geroprotection

This term refers to drugs or interventions that target the fundamental biological hallmarks of aging, such as decreased autophagy, increased senescence, or genomic instability, with the aim of protecting against or delaying the aging process itself, rather than specific age-related diseases.

Informative Censoring

A methodological limitation in some studies where participants who deviate from the initial inclusion criteria (e.g., stopping a drug, progressing to a more severe condition) are excluded from the final analysis. This can bias results by only considering a subset of the original group.

Hazard Ratio

A statistical measure used in survival analysis to compare the risk of an event (like death) between two groups over time. A hazard ratio greater than 1 indicates an increased risk in the exposed group, while less than 1 indicates a reduced risk.

Statistical Power

The probability that a study will find a statistically significant difference between groups if a real difference truly exists. It is crucial for interpreting negative results, as a study might fail to find significance if it is underpowered to detect the actual effect size.

Kaplan-Meier Curve

A graphical representation of survival probability over time, commonly used in medical research to estimate the fraction of patients living for a certain amount of time after treatment or diagnosis. The curve always decreases monotonically, showing cumulative survival.

Belief Effects

These are physiological and psychological changes in response to a treatment that are influenced by an individual's knowledge, expectations, and beliefs about that treatment. Unlike binary placebo effects, belief effects can scale with the amount and type of information received, impacting biological responses.

Nicotinic Receptors

These are a type of acetylcholine receptor found in the brain, particularly in areas like the thalamus and basal forebrain. When activated, they increase the signal-to-noise ratio of sensory information, leading to enhanced focus and clarity.

?
What is metformin and how does it work?

Metformin is a generic drug used for type 2 diabetes that inhibits complex one of the mitochondria, which reduces the liver's glucose output and helps lower blood sugar.

?
What is insulin resistance and what causes it?

Insulin resistance is when cells require more insulin to take up glucose, often caused by intramuscular fat accumulation, physical inactivity, sleep deprivation, and high cortisol levels.

?
What was the key finding of the 2014 Bannister study on metformin and longevity?

The Bannister study found that type 2 diabetics on metformin had a 15% relative reduction in all-cause mortality over 2.8 years compared to matched non-diabetic controls, suggesting a potential geroprotective effect.

?
How did the 2022 Keys et al. study re-evaluate metformin's longevity benefits?

The Keys et al. study, using a larger Danish registry and a more rigorous analysis including discordant twins and different censoring methods, found that diabetics on metformin still had a significantly higher mortality risk compared to non-diabetic controls, contradicting Bannister's finding of a survival advantage.

?
Why is it difficult to draw definitive conclusions from epidemiological studies?

Epidemiological studies, despite large sample sizes, cannot fully control for all confounding variables and inherent biases, making it hard to isolate the effect of a single intervention without randomized assignment.

?
Does Peter Attia still take metformin for longevity?

No, Peter Attia stopped taking metformin five years ago, partly due to concerns about the Bannister study's methodology and its observed impact on his resting lactate levels and exercise performance.

?
Is there a longevity benefit to short periods of caloric restriction or fasting?

Peter Attia believes there is no clear evidence that short-term caloric restriction or fasting offers a direct longevity benefit to humans, especially considering potential muscle mass loss and the lack of reliable aging biomarkers.

?
How do 'belief effects' differ from 'placebo effects'?

Placebo effects are binary (drug vs. no drug), while belief effects involve the scaling of physiological and psychological responses based on the amount and type of information an individual believes about a treatment, even if the actual dose is constant.

?
How does nicotine affect the brain to improve cognitive performance?

Nicotine, by binding to nicotinic acetylcholine receptors in areas like the thalamus and basal forebrain, increases the signal-to-noise ratio of sensory information, enhancing focus and clarity.

?
What are the implications of belief-dependent drug responses for medicine?

The study suggests that what patients are told and believe about a drug's dose and effects can significantly alter their brain's physiological response, potentially influencing treatment outcomes and even how side effects are experienced.

1. Leverage Belief Effects

Recognize that your beliefs about drug doses and effects can biologically impact your physiology and brain activation, not just subjectively.

2. Deep Self-Analysis

Engage in intensive analysis, potentially with a skilled professional, to explore your subconscious and catalyze transformative life decisions and experiences.

3. Exercise for Insulin Sensitivity

Prioritize regular exercise as it is one of the most important actions to ward off insulin resistance and maintain metabolic health.

4. Sleep for Insulin Sensitivity

Ensure adequate sleep, as sleep deprivation can profoundly reduce glucose disposal and induce insulin resistance.

5. Prevent Excess Fat Accumulation

Avoid accumulating excess fat, especially its spillover into muscles, liver, and pancreas, as this exacerbates insulin resistance.

6. Maintain Glucose Homeostasis Through Lifestyle

Achieve glucose homeostasis and energy balance through good sleep hygiene, consistent exercise, and thoughtful eating, without necessarily relying on caloric deficit.

7. Reframe Stress Positively

Reframe your perception of stress by believing it enhances performance, which can lead to improved outcomes compared to viewing it as limiting.

8. Reframe Drug Side Effects

View drug side effects as evidence that the medication is working, which can make the experience less negative and potentially increase perceived relief from primary symptoms.

9. Dosage Belief Alters Response

Understand that your belief about a drug’s dose can significantly alter your brain’s physiological response, influencing how your body reacts.

10. Influence Satiety Through Belief

Recognize that your belief about a food’s caloric content and nutritional value can influence physiological responses like ghrelin suppression and satiety.

11. Believe in Exercise Benefits

Actively acknowledge and believe in the health benefits of your daily physical activity, as this belief alone can lead to greater positive physiological outcomes like weight loss.

12. Avoid Nocebo Effect

Be cautious about dwelling on potential negative side effects of a drug, as this can trigger the nocebo effect, leading to the subjective experience of those side effects.

13. Structured Paper Reading

When reading scientific papers, identify the questions, understand the approach, note the findings, and critically assess if conclusions are substantiated by the data.

14. Research Scientific Methods

When encountering unfamiliar scientific methods in a paper, look up their purpose online to significantly aid comprehension.

15. Prioritize Figures in Papers

After reading the title and abstract, first review the figures to digest visual information before delving into the main text for deeper understanding.

16. Adapt Paper Reading Strategy

Adjust your paper reading approach based on familiarity; for new topics, use figures to generate questions, then read methods for clarity.

17. Review Supplemental Paper Info

Always check the supplemental information of scientific papers, as crucial data or analyses are often placed there due to journal length constraints.

18. Start a Journal Club

Engage in or resume a formal journal club (e.g., monthly) to review scientific papers, as it’s a great way to learn and develop interpretation skills.

19. Interpret Scientific Papers

Develop the skill of reading and interpreting scientific papers to understand research findings and think like scientists and clinicians.

20. Don’t Rely Solely on Abstracts

Avoid relying solely on a paper’s abstract for full understanding, as it’s usually insufficient to grasp the complete findings and implications.

21. Titrate Drug Dosages

When starting a new medication, titrate the dose up gradually rather than starting at a full dose to avoid severe side effects.

22. Manage Cortisol Levels

Be aware that hypercortisolemia (high cortisol) can contribute to insulin resistance, implying a need to manage stress or other factors that elevate cortisol.

23. Re-evaluate Metformin for Longevity

Consider that metformin has not shown success in the ITP for geroprotection, suggesting re-evaluation of its use solely for anti-aging purposes in insulin-sensitive individuals.

24. Monitor Resting Lactate

If taking a weak mitochondrial inhibitor, monitor resting fasted lactate levels (aim for 0.3-0.6 millimole) as elevated levels may indicate increased shunting of glucose to lactate.

25. Metformin & Muscle Growth

Be aware that metformin may attenuate hypertrophy and strength gains, which could be a factor for individuals prioritizing muscle mass and exercise performance.

26. Explore SGLT2 Inhibitors

Consider SGLT2 inhibitors as a potential drug for glucose disposal, based on mechanistic studies, ITP results, and human trials showing benefits even in non-diabetics.

27. Eat Unprocessed Foods

Focus on consuming most nutrients from unprocessed or minimally processed food sources, as this approach can enhance satiety and allow for greater food intake.

28. Acknowledge Longevity Biomarker Gaps

Understand that current longevity interventions lack reliable biomarkers to indicate whether they are effectively moving one in the right direction or at the correct dosage.

29. Gradual Nicotine Reduction

When attempting to quit nicotine, consider progressively reducing the actual dose while maintaining the belief that the dose is consistent to mitigate reward pathway disruption.

The difference between being metabolically healthy and having profound type 2 diabetes is one teaspoon of glucose in your bloodstream.

Peter Attia

If a 95% confidence interval does not cross the number zero, it's statistically significant.

Peter Attia

What we believe about the effects of a drug, presumably, in addition to what we believe about how much we're taking and what those effects ought to be, clearly are impacting at least the way that our brain reacts to, to those drugs.

Andrew Huberman

Your brain is a prediction making machine. It's a data interpretation machine, but it's clear that one of the more important pieces of data are your beliefs about how these things impact you.

Andrew Huberman

Anal seepage, only a physician... could find it, a, um, an appropriate term to describe.

Andrew Huberman

Process for Reading Scientific Papers

Andrew Huberman
  1. Identify the general and specific questions the paper is asking.
  2. Understand the approach and methods used to test those questions, looking up unfamiliar terms.
  3. Determine what the researchers found, often by reading figure headers first.
  4. Go back at the end and ask whether the major conclusions drawn in the paper are truly substantiated by what was found and what was done.
5 grams
Glucose in total circulation (normal blood sugar) In a metabolically healthy person
1 teaspoon
Difference in glucose level between metabolically healthy and profound type 2 diabetes Consistently elevated glucose in the bloodstream
75 to 80%
Percentage of glucose stored in muscles Muscle is responsible for most glucose disposal
6 years
Average life shortening due to type 2 diabetes Actuarial difference for all comers with type 2 diabetes
14.4 deaths per thousand patient years
Bannister study (2014) crude death rate for metformin group Type 2 diabetics on metformin, with informative censoring
15.2 deaths per thousand patient years
Bannister study (2014) crude death rate for control group Matched non-diabetic controls
15%
Bannister study (2014) relative reduction in all-cause death for metformin group Over 2.8 years median follow-up, compared to controls
Half a million people
Keys et al. study (2022) sample size Sampled from a Danish health registry
1.48
Keys et al. study (2022) hazard ratio for singletons (unadjusted) Metformin group had 48% greater chance of dying than non-diabetic counterparts
1.32
Keys et al. study (2022) hazard ratio for singletons (adjusted for meds/marital status) Metformin group had 32% greater chance of dying than non-diabetic counterparts
2.15
Keys et al. study (2022) hazard ratio for twins (unadjusted) Diabetic twin on metformin had 115% greater chance of dying than non-diabetic co-twin
1.70
Keys et al. study (2022) hazard ratio for twins (adjusted for meds/marital status) Diabetic twin on metformin had 70% greater chance of dying than non-diabetic co-twin
About half
Reduction in glucose disposal due to sleep deprivation After sleeping only 4 hours for a week
15%
Rapamycin lifespan extension in mice (ITP) Even when given very late in life (60-month-old mice)