Iñigo San Millán, Ph.D.: Zone 2 Training and Metabolic Health (Ep. #85 Rebroadcast)

Dec 27, 2021 Episode Page ↗
Overview

Dr. Iñigo San Millán, Assistant Professor at the University of Colorado School of Medicine, joins Peter Attia to discuss mitochondrial function, energy systems, and the six training zones. They extensively cover zone 2 training, lactate as a crucial fuel, and its role in metabolic health and exercise.

At a Glance
13 Insights
2h 50m Duration
16 Topics
9 Concepts

Deep Dive Analysis

Iñigo San Millán's Athletic Background and Career Shift

Understanding Aerobic and Anaerobic Energy Systems

Iñigo's Six Zones of Exercise Training

Lactate: A Crucial Fuel, Not Just Waste Product

Zone 2 Training: Physiology, Fuel, and Lactate

Assessing Mitochondrial Function via Zone 2 Lactate

Fat Oxidation Capacity as a Mitochondrial Health Marker

Metabolic Differences: Elite Athletes vs. Diabetics

Physiology of Zone 3, Zone 4, and Lactate Threshold

Dietary Fueling Strategies and Glycolytic Function

Exercise, Insulin Sensitivity, and Type 1 Diabetes

Metformin's Effect on Exercise and Mitochondrial Function

The Concept and Risks of "Double Diabetes"

Optimal Dosing and Balancing Zone 2 Training

Lactate's Role in the Warburg Effect and Cancer

Doping and Altitude Training in Professional Cycling

Aerobic Metabolism

Aerobic metabolism refers to the majority of physical activity where ATP is generated at a rate slow enough that all metabolic demands can be met through mitochondrial oxidation, primarily of fatty acids and glucose. It occurs in the presence of oxygen, but the key is the rate of ATP demand being met by mitochondrial capacity.

Anaerobic Metabolism

Anaerobic metabolism occurs when the demand for ATP exceeds the capacity of the mitochondria and even the cytosolic production of ATP. This forces the body to use ATP already stored in the muscles, such as during a sprint, and is characterized by a rapid, but quickly fatiguing, energy supply.

Lactate as a Fuel/Signaling Molecule

Lactate is a mandatory byproduct of glucose utilization and is considered a crucial fuel for the body, including the brain and heart. It also acts as a major signaling molecule that helps maintain homeostasis in various metabolic pathways throughout the body.

Zone 2 Training

Zone 2 training is an exercise intensity that maximally stimulates slow-twitch muscle fibers without recruiting fast-twitch fibers. It coincides with the 'fat max,' where the highest amount of fat is oxidized, and is considered optimal for improving mitochondrial function, fat burning, and lactate clearance capacity.

Fat Max

Fat max is the specific exercise intensity at which an individual oxidizes the highest amount of fat per minute. This physiological point typically occurs within Zone 2 training and is a key indicator of metabolic efficiency and mitochondrial health.

Respiratory Quotient (RQ) / Respiratory Exchange Ratio (RER)

RQ/RER is the ratio of carbon dioxide produced to oxygen consumed, measured from expired gases. A ratio of 0.7 indicates almost exclusive fat oxidation, while a ratio of 1.0 indicates exclusive glucose oxidation, serving as an indirect measure of fuel utilization.

Athlete's Paradox (Intramuscular Triglycerides)

This paradox describes how fat droplets found near mitochondria in muscle cells are associated with insulin resistance in unhealthy individuals, but in elite athletes, they represent an active, readily utilized reservoir of fuel. In athletes, these fat droplets are efficiently oxidized by mitochondria, contributing significantly to energy production.

Non-Insulin Dependent Glucose Uptake

This refers to the process where muscle contraction directly stimulates the translocation of GLUT4 glucose transporters to the muscle cell surface, allowing glucose to enter the cell without the need for insulin. This mechanism is particularly beneficial for individuals with insulin resistance and is enhanced by exercise.

Warburg Effect

The Warburg Effect describes the metabolic characteristic of cancer cells, which consume large amounts of glucose and produce significant lactate, even in the presence of oxygen. This lactate is not merely a byproduct but also acts as a signaling molecule that promotes various aspects of carcinogenesis, including proliferation and metastasis.

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What are the main energy systems used during exercise?

The body primarily uses aerobic metabolism for most activities, even at higher intensities, with anaerobic metabolism reserved for very short, maximal efforts. The main fuels are fatty acids and glucose, which are oxidized in different muscle fiber types.

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What is lactate and why is it important?

Lactate is a mandatory byproduct of glucose utilization, not a waste product, and is a crucial fuel for the brain, heart, and other cells. It also serves as a major signaling molecule that helps maintain metabolic homeostasis throughout the body.

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What is Zone 2 training and why is it beneficial?

Zone 2 training is an exercise intensity that fully stimulates slow-twitch muscle fibers and aligns with the 'fat max,' where fat oxidation is highest. It is considered the optimal intensity for improving mitochondrial function, enhancing fat burning, and increasing lactate clearance capacity.

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How can blood lactate levels assess mitochondrial function?

Measuring blood lactate levels during exercise, particularly at the Zone 2 threshold, provides an indirect assessment of mitochondrial function. Higher lactate accumulation indicates that the muscle's mitochondria are less efficient at clearing or metabolizing lactate.

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How does the body's ability to oxidize fat relate to metabolic health?

A poor capacity to oxidize fat, especially at rest or low exercise intensities, is a significant indicator of mitochondrial dysfunction and is commonly seen in conditions like type 2 diabetes. Conversely, high fat oxidation capacity signifies robust mitochondrial health.

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What is the 'athlete's paradox' regarding intramuscular triglycerides?

The 'athlete's paradox' highlights that while intramuscular fat droplets are linked to insulin resistance in unhealthy individuals, in elite athletes, these fat deposits are highly active fuel reservoirs. These active fat droplets are efficiently oxidized by mitochondria, contributing significantly to energy during exercise.

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How does exercise improve glucose uptake without insulin?

Muscle contraction itself triggers pathways that translocate GLUT4 glucose transporters to the muscle cell surface, enabling glucose to enter cells independently of insulin. This non-insulin dependent glucose uptake is a key benefit of exercise, particularly for improving insulin sensitivity.

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What is 'double diabetes'?

Double diabetes refers to individuals with type 1 diabetes who also develop insulin resistance or other characteristics of type 2 diabetes. This is a growing concern, especially given the high prevalence of type 2 diabetes and pre-diabetes in the general population.

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What is the minimum effective dose for Zone 2 training?

For maintaining mitochondrial function, two days a week of Zone 2 training is generally sufficient. To significantly improve function, three to five days a week, for about one to one and a half hours per session, is recommended, especially for individuals with metabolic dysfunction.

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How does metformin impact exercise benefits and lactate levels?

Metformin can increase lactate levels, possibly by increasing glucose flux into cells or inhibiting mitochondrial function (specifically complex I). Some research suggests it might blunt exercise benefits, particularly in metabolically healthy individuals, by affecting mitochondrial efficiency.

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What is the Warburg Effect in cancer and lactate's role?

The Warburg Effect describes cancer cells' tendency to consume large amounts of glucose and produce significant lactate, even with sufficient oxygen. Lactate is not just a byproduct but acts as a signaling molecule that drives various aspects of carcinogenesis, including cell proliferation, angiogenesis, and metastasis.

1. Maximize Mitochondrial Function

Engage in Zone 2 exercise, which stimulates slow-twitch muscle fibers to their fullest and maximizes fat oxidation, leading to significant improvements in mitochondrial function and lactate clearance capacity.

2. Identify Zone 2 by Conversation

To gauge Zone 2 intensity without equipment, exercise at the highest level of exertion where you can still comfortably carry on a conversation, indicating optimal aerobic output.

3. Zone 2 Lactate Levels

Aim for lactate levels between 1.5 to 2 millimoles during Zone 2 training, as this range signifies efficient fat oxidation and maximal mitochondrial output without significant lactate accumulation.

4. Zone 2 Training Frequency

To maintain mitochondrial function, aim for at least two days of Zone 2 training per week; to significantly improve it, increase frequency to five days a week.

5. Prioritize Mitochondrial Conditioning

Approach exercise not just as calorie burning, but as ‘mitochondrial conditioning’ or ‘reprogramming’ to enhance fuel partitioning and overall metabolic health.

6. Zone 2 Dose for Diabetics

For individuals with pre-type 2 or type 2 diabetes, aim for one to one and a half hours of Zone 2 training, four days a week, to effectively reverse metabolic dysfunction.

7. Implement Post-Exercise Cool Down

After high-intensity exercise, perform a cool-down period to help reduce post-exercise hyperglycemia, potentially eliminating the need for insulin correction in diabetic individuals.

8. Exercise Immediately After Meals

For individuals with type 2 diabetes, exercise immediately after eating to leverage muscle contraction for non-insulin dependent glucose uptake, reducing the need for insulin.

9. Adjust Metformin for Zone 2

If taking metformin and performing Zone 2 exercise, consider adjusting the timing or dose (e.g., stopping it the night before) to prevent potential blunting of exercise benefits and reduce elevated lactate levels, as observed in personal experimentation and suggested by recent papers.

10. Adopt Sustainable Health Habits

When choosing health and exercise routines, prioritize practices that you can realistically maintain for the rest of your life, rather than unsustainable extreme measures.

11. Consider Carbohydrate Restriction

For metabolically unhealthy individuals not aiming for elite athletic performance, carbohydrate restriction can be a powerful tool to improve metabolic health.

12. Incorporate Varied Fasting

Implement different types of fasting (e.g., weekly, monthly, quarterly) as a strategy to maintain metabolic balance and sustain long-term health habits.

13. Reduce Insulin Before Exercise (Type 1)

Type 1 diabetics, under clinical guidance, should consider reducing their insulin dose before exercise, as muscle contraction increases non-insulin dependent glucose uptake and insulin sensitivity, helping prevent hypoglycemia.

Everybody sort of has a sense that all roads point to the mitochondria.

Peter Attia

In the elite athletes have the perfect metabolism. And mitochondria is at the epicenter of metabolism and health.

Iñigo San Millán

Lactate is the most important, if not the most important fuel for the body.

Iñigo San Millán

Watts is a mechanical parameter, but heart rate is a physiological parameter and responds to the physiological metabolic stress.

Iñigo San Millán

If you can't do it for the rest of your life, you have to come back to the, why am I doing this?

Peter Attia

The most potent drugs we have are food and exercise.

Peter Attia

There's no population on earth who has as many carbohydrates and simple sugars as these athletes by a landslide.

Iñigo San Millán

Zone 2 Training for Mitochondrial Improvement

Iñigo San Millán
  1. Perform Zone 2 training 3-5 days per week (2 days for maintenance).
  2. Each session should last 1 to 1.5 hours.
  3. Maintain an intensity where lactate levels are between 1.5-2 millimoles, or at a level where you can still comfortably carry on a conversation.

Cool-down Protocol for Post-Exercise Hyperglycemia

Iñigo San Millán
  1. After high-intensity exercise, perform a cool-down period.
  2. This helps reduce post-exercise hyperglycemia by continuing muscle contraction, which facilitates non-insulin dependent glucose uptake.
0.7 to 1 millimol
Resting lactate levels In a healthy individual
10-12 millimoles
Maximal lactate levels In a normal person during a violent protocol
15-17 millimoles (up to 20 millimoles)
Maximal lactate levels In a world-class rower during a maximal effort
1.5 to 2 millimoles
Zone 2 lactate levels Slightly above resting levels, indicating a steady state without net accumulation
300 watts (just below 5 watts per kilogram)
Zone 2 power output For a world-class cyclist weighing 60-65 kg
200 watts
Zone 2 power output For a reasonably fit 'weekend warrior'
120 watts (about 1.5 watts per kilogram)
Zone 2 power output For a person with type 2 diabetes
3 to 4 times higher
Mitochondrial content and size In well-trained athletes compared to less fit individuals
85-100 (on a 0-100 scale)
Glycogen storage capacity For world-class athletes, measured by high-frequency ultrasound
60-70 (on a 0-100 scale)
Glycogen storage capacity For moderately active individuals, measured by high-frequency ultrasound
30-50 (on a 0-100 scale)
Glycogen storage capacity For individuals with type 2 diabetes, measured by high-frequency ultrasound
25% to 35%
Fat oxidation from intramuscular triglycerides Contribution to total fat oxidation during exercise in elite athletes
850 grams (12 grams per kilogram of body weight)
Daily carbohydrate intake For a Tour de France cyclist (e.g., 70 kg athlete)
1500 calories
Daily simple carbohydrate intake For a Tour de France cyclist, representing 30-50% of total carbohydrate intake
50%
Insulin secretion decrease during exercise In regular people, due to non-insulin dependent glucose uptake
50%
Prevalence of pre-diabetes or diabetes Of the U.S. adult population
Two-thirds
Insulin sales for type 2 diabetics Of all insulin sold in America
5% to 10%
Risk of muscle symptoms from statins Reported in published literature
4%
Increased risk of diabetes from statins Long-term risk
10 millimoles (up to 40 millimoles)
Lactate levels in cancer microenvironment Compared to normal physiological levels
2 to 8-fold
Overexpression of oncogenes by lactate In MCF7 breast cancer cells compared to control