How Cannabis Impacts Health & the Potential Risks | Dr. Matthew Hill

Episode 184 Jul 8, 2024 Episode Page ↗
Overview

Dr. Matthew Hill, a professor at the University of Calgary, discusses cannabis biology, including THC vs. CBD effects, routes of administration, and the link between cannabis use and mental health conditions like psychosis and anxiety. He clarifies common misconceptions and potential benefits and risks.

At a Glance
19 Insights
3h 52m Duration
27 Topics
9 Concepts

Deep Dive Analysis

Introduction to Cannabis, THC, CBD, and Terpenes

Describing the Psychoactive Effects and Cannabis 'High'

Cannabis and Brain Function: CB1 Receptor and Endocannabinoids

Types of Endocannabinoids: Anandamide and 2-AG

The 'Munchies': Cannabis and Appetite Stimulation

THC Pharmacology and Comparison to Anandamide

THC, CB1 Receptors, and Intoxication Mechanisms

Cannabis Effects on Focus and Memory

Routes of Cannabis Administration and Concentration

Self-Regulation of THC Intake and Concentrates

Addiction and Cannabis Use Disorder

Cannabis Legalization, Use Trends, and Edible-Related ER Visits

Oral Consumption of Edibles: Dosing and Time Course

Cannabis and Drug Testing: Detection and Influencing Factors

Cannabis and Hormones: Testosterone, Estrogen, and Sperm Quality

Cannabis Use During Pregnancy

Vaping Cannabis Products

Cannabis, Psychosis, and Anxiety

Cannabis, Psychosis, Schizophrenia, and Genetics

Cannabis Use and Schizophrenia: Causality vs. Predisposition

Cannabis Strains: Indica, Sativa, and Expectancy Bias

CBD: Pediatric Epilepsy, Adenosine, and Efficacy

Entourage Effect, Placebo Effect, and CBD Doses

Cannabis Health Risks: Cardiovascular and Other Concerns

Cannabinoid Hyperemesis Syndrome (Cyclic Vomiting Syndrome)

Potential Cannabis Benefits: Pain, Stress, Anxiety, and PTSD

Cannabis and Anxiety: Anandamide and Stress Response

Cannabinoid Type 1 (CB1) Receptor

The most widely expressed receptor in the brain, primarily mediating the psychoactive effects of THC. Its physiological role is to maintain homeostasis by regulating neurotransmitter release, acting as a circuit breaker to prevent runaway excitation or inhibition of neurons.

Endocannabinoids

Molecules naturally produced by the body, such as anandamide and 2-AG, that bind to cannabinoid receptors. They are unique in that they are released from the postsynaptic neuron and travel backward to the presynaptic neuron to regulate neurotransmitter release, thereby maintaining neural homeostasis.

Retrograde Signaling

A distinct form of neurotransmission where endocannabinoids are produced by the postsynaptic neuron and then travel backward across the synapse to act on the presynaptic neuron. This mechanism allows for fine-tuned regulation of how much neurotransmitter is released by the presynaptic cell.

Anandamide

One of the two primary endocannabinoids, named from the Sanskrit word for bliss. It is characterized as a high-affinity, low-efficacy molecule that may play a tonic role in setting the steady state of brain circuits, contributing to baseline neural regulation.

2-Arachidonylglycerol (2-AG)

The second primary endocannabinoid, which has lower affinity but higher efficacy at the CB1 receptor compared to anandamide. It is thought to act in a phasic, on-demand manner, being rapidly produced during significant neural activity to powerfully adjust circuit function.

Cannabis Use Disorder

A problematic pattern of cannabis use leading to clinically significant impairment or distress, with diagnostic criteria similar to other substance use disorders. It can manifest as preoccupation with cannabis, failure to meet responsibilities, and withdrawal symptoms upon cessation.

Expectancy Bias

A psychological phenomenon where an individual's expectations about a substance's effects significantly influence their subjective experience. In cannabis use, what a person is told about a particular strain (e.g., 'sativa for energy') often strongly predicts the effects they report feeling.

Entourage Effect

A theoretical concept suggesting that the various compounds in the cannabis plant (THC, CBD, terpenes, minor cannabinoids) interact synergistically. This interaction is hypothesized to produce a combined effect that is different from, or greater than, the sum of their individual effects when isolated.

Cannabinoid Hyperemesis Syndrome (Cyclic Vomiting Syndrome)

A rare but real condition observed in some heavy, chronic cannabis users, characterized by intractable, recurring episodes of severe nausea and vomiting. Bizarrely, a hot shower or capsaicin cream has been reported to alleviate its symptoms, suggesting a link to thermoregulation or autonomic balance.

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What are the main psychoactive components of cannabis?

The primary psychoactive component is delta-9 tetrahydrocannabinol (THC), which dictates the intoxicating and psychoactive properties. Cannabidiol (CBD) is another prominent cannabinoid, but it is not intoxicating.

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How does THC produce its effects in the brain?

THC primarily acts on cannabinoid type 1 (CB1) receptors, which are widely expressed throughout the brain. Unlike the body's own endocannabinoids that act in a targeted way, ingested THC indiscriminately activates these receptors across many brain networks, altering information processing and perception.

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Why does cannabis cause 'the munchies' or stimulate appetite?

THC activates CB1 receptors in feeding circuits in the hypothalamus and reward circuitry, mimicking the brain's response to fasting. This enhances the reward value of food and overrides satiety signals, leading to increased food-seeking and consumption, particularly of high-calorie foods.

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Can people develop a cannabis use disorder?

Yes, people can develop cannabis use disorder, which is defined by criteria similar to other substance use disorders, involving preoccupation with the drug, inability to meet responsibilities, and withdrawal symptoms if use is stopped. For weekly users, the propensity for developing a use disorder is around 30%.

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How do different routes of cannabis administration (smoking vs. edibles) affect the experience?

Inhaling cannabis (smoking/vaping plant matter) leads to rapid onset (2-5 minutes) and shorter duration (2-4 hours) of effects due to quick absorption. Edibles have a delayed onset (30-90 minutes) and longer duration (4-8 hours) because THC undergoes first-pass metabolism in the liver, producing a more potent metabolite (11-hydroxy-THC) that accumulates slowly.

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Does cannabis cause schizophrenia?

While there is a strong statistical association between cannabis use, especially in adolescence, and the development of schizophrenia, establishing direct causality is challenging. The prevailing view is that cannabis can accelerate the onset and worsen the prognosis of schizophrenia in individuals already genetically or biologically predisposed, rather than causing it de novo.

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Are there real differences in effects between 'Indica' and 'Sativa' cannabis strains?

The terms Indica and Sativa are botanical classifications that do not reliably track with chemical composition or subjective effects. Research suggests that the variability within these classifications is greater than between them, and subjective differences are largely attributed to expectancy bias.

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What are the established medical benefits of CBD?

CBD has been clearly shown to be effective in treating specific forms of pediatric epilepsy, such as Dravet syndrome, at very high doses (e.g., 1,500-2,000 mg). However, for other conditions like sleep, pain, or anxiety, there is little conclusive clinical trial data to support significant benefits at the much lower doses typically found in commercial products.

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What are the main health risks associated with cannabis use?

Key health risks include potential lung damage from smoking, possible cardiovascular issues (like tachycardia and vascular stiffness), and a rare but real cyclic vomiting syndrome in heavy users. Cannabis use is contraindicated for individuals with a predisposition to schizophrenia or bipolar disorder.

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Can cannabis help with chronic pain?

Cannabis has some mild analgesic properties, but its primary benefit for chronic pain patients seems to be stripping away the emotional component of pain, making it feel like 'background noise.' This allows patients to sleep better and function more effectively, even if the physical sensation of pain persists.

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Can cannabis help with anxiety?

Many people report using cannabis to reduce anxiety, and some animal and limited human studies suggest lower doses can be anxiolytic. However, higher doses can paradoxically increase anxiety, and its efficacy in clinical trials for anxiety disorders is still being investigated.

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Can cannabis help with PTSD-related nightmares?

Anecdotal reports and small clinical studies suggest that cannabis, particularly THC, can effectively suppress nightmares in individuals with PTSD, which is a significant benefit for improving sleep quality and overall well-being. This may be due to its effects on REM sleep architecture.

1. Avoid Cannabis with Psychotic Risk

Individuals diagnosed with schizophrenia or bipolar disorder, or those with a first-degree family member having these conditions, should avoid cannabis to prevent triggering or accelerating disease onset and worsening long-term prognosis.

2. Avoid Cannabis with Cardiovascular Issues

Individuals with pre-existing cardiovascular issues should avoid cannabis due to potential risks like tachycardia and vascular effects, which could unmask vulnerabilities or trigger events.

3. Cease Cannabis for Fertility Issues

If struggling to conceive, both males and females should consider cutting out cannabis use, as it may negatively impact sperm quality or overall fertility in some individuals.

4. Avoid Cannabis During Pregnancy

Most people stop cannabis use upon learning of pregnancy; it is advisable to avoid cannabis during pregnancy due to unknown effects on the developing fetus and potential legal ramifications if accidental pediatric ingestion occurs.

5. Secure Edibles from Children

Store THC-containing edibles securely away from children, as accidental ingestion can lead to severe intoxication, emergency room visits, and potential legal consequences for parents.

6. Practice “Low and Slow” Edible Dosing

When consuming cannabis edibles, start with a very low dose (e.g., 2.5-5mg THC) and wait at least 90 minutes for effects to manifest before considering additional intake, as onset is slow and effects can be potent and long-lasting.

7. Exercise Caution with Concentrates

Be extremely cautious with cannabis concentrates (e.g., dabs, distillates) as they can reach 90-98% THC, are difficult to titrate, and can lead to blood THC levels double to triple that of high-potency flower, increasing risks of adverse effects and tolerance.

8. Do Not Increase During Paranoia

If experiencing anxiety or paranoia from cannabis, do not consume more, as this will likely exacerbate the negative experience.

9. Prioritize Unprocessed Whole Foods

Consume the majority of your diet from unprocessed or minimally processed sources to ensure adequate intake of vitamins, minerals, probiotics, and micronutrients, supporting physical and mental health and preventing overeating.

10. Prioritize Electrolyte Hydration

Consume adequate electrolytes (sodium, magnesium, potassium) to prevent diminished cognitive and physical performance, especially upon waking and during physical exercise or hot days when sweating.

11. Control Sleep Environment Temperature

To fall and stay deeply asleep, and to wake up refreshed, ensure your body temperature drops by 1-3 degrees and then increases by 1-3 degrees, which can be achieved by controlling your sleeping environment’s temperature.

12. Seek Expert Online Therapy

Engage in therapy with a licensed therapist to gain insights into emotional and relationship life, understand oneself better, and inspire positive action, ensuring good rapport, emotional support, and directed guidance.

13. Titrate Inhaled Cannabis Carefully

When inhaling cannabis, especially higher potency flower, take small amounts and allow a few minutes for effects to manifest to avoid overconsumption and adverse responses.

14. Consider Edibles for Lung Health

To mitigate risks of lung damage, emphysema, and other issues associated with combustion from smoking cannabis, consider oral routes of administration (edibles).

15. Vaporize Plant Matter to Reduce Harms

If consuming cannabis via inhalation, consider vaporizing plant matter using a device that heats it without combustion, as this avoids harmful combustion byproducts found in smoking.

16. Caution with Oil-Based Vaping

Be cautious with oil-based cannabis vaping products, as their long-term health effects and specific chemical compositions are not well-researched or regulated, potentially leading to unforeseen harms.

17. Avoid Exercise Before Drug Test

If facing a cannabis drug test, avoid intense exercise or weight loss efforts beforehand, as these can release stored THC from fat cells back into the bloodstream, potentially causing a positive test even after abstinence.

18. Skepticism for Low-Dose CBD

Be aware that most reported effects from low-dose CBD products (e.g., 10-20mg) are likely placebo effects, as efficacious clinical doses for conditions like pediatric epilepsy are typically 1500-2000mg, and oral CBD has very poor bioavailability (around 4%).

19. Hot Shower for Cyclic Vomiting

If experiencing cannabis-induced cyclic vomiting syndrome, a hot shower or capsaicin cream application may provide relief, suggesting an interaction with thermoregulation or autonomic balance.

The main role of endocannabinoids is really to regulate plasticity or homeostasis, allow flexibility of circuits to either goose up their activity or ramp it down if they need to, depending on the environment, depending on the experience of the organism.

Dr. Matthew Hill

THC is actually only a partial agonist. It's not even a full agonist at CB1. But it is high affinity.

Dr. Matthew Hill

The problem is when you just blanket activate all the CB1 receptors in the brain indiscriminately, like you do when you consume cannabis with THC, the resulting effect is the intoxicating state.

Dr. Matthew Hill

If someone has schizophrenia, cannabis is contraindicated. Like, you shouldn't be using cannabis if you have schizophrenia.

Dr. Matthew Hill

To me, cannabis is fuel on a fire. So if someone is prone to developing schizophrenia, adding cannabis into the mix, I think will make it kick in faster and harder.

Dr. Matthew Hill

The biggest predictor of what someone feels when they consume cannabis is what they're told on the label it's going to do to them.

Dr. Matthew Hill

I would argue that the overwhelming majority of the effects of CBD that people report are all placebo effects.

Dr. Matthew Hill

My pain's still there, but now the pain's background noise. So I can sleep at night, and just being able to sleep, I think, is actually providing a huge amount of the benefit to that community.

Dr. Matthew Hill
5%
Typical THC concentration in cannabis from the 1970s Compared to modern cannabis, which is significantly higher.
20-30%
Typical THC concentration in modern commercial cannabis flower Botanically, cannabis plants generally cannot exceed this range.
Up to 90-98%
THC concentration in cannabis concentrates (e.g., dabs, distillates) These are highly potent products that are difficult to titrate.
Around 100 nanograms per ml
Typical blood THC levels achieved by self-titrating inhaled cannabis Observed in both NIDA-sourced cannabis studies and 'Canavan' ecological studies, regardless of initial plant potency.
200-300 nanograms per ml
Typical blood THC levels from high-potency concentrates Significantly higher than inhaled flower, indicating difficulty in self-titration.
2-5 minutes
Onset time for psychoactive effects after inhaling cannabis Allows for better self-titration compared to edibles.
15-30 minutes
Duration of peak intoxication from inhaled cannabis The most intense effects occur within this timeframe.
2-4 hours
Overall duration of intoxication from inhaled cannabis The bulk of intoxication is typically over within two hours, with some residual effects lasting longer.
30-90 minutes
Onset time for psychoactive effects after orally consuming edibles This delay often leads to accidental overconsumption.
4-8 hours
Duration of intoxication from orally consumed edibles Effects last significantly longer due to first-pass metabolism and metabolite accumulation.
10 milligrams
Maximum THC content per edible package in Canada This limit is set by regulation to mitigate risks of overconsumption.
5-10 milligrams
Typical intoxicating dose of edibles for relatively naive or intermittent users Many individuals will feel effects at 5mg, and almost everyone at 10mg.
2-5 nanograms per ml
Typical blood THC levels from edibles Significantly lower than inhaled cannabis, yet still produces profound intoxication due to 11-hydroxy-THC metabolite.
30 days
Recommended abstinence period for passing a cannabis drug test THC is lipophilic and can store in fat, slowly leaking back into the bloodstream; highly variable based on usage and individual.
Approximately 4%
Bioavailability of orally consumed CBD (standard conditions) Very little CBD is absorbed into the bloodstream when taken orally.
Up to 20%
Enhanced bioavailability of orally consumed CBD (with fatty meal) Fat in the stomach and gut can significantly improve CBD absorption.
1,500-2,000 milligrams
Clinical dose range of CBD for pediatric epilepsy studies These high doses are vastly different from typical commercial CBD products.
2-25 milligrams
Common dose range of commercial CBD products (e.g., gummies) These doses are generally considered too low to have significant biological activity based on current research.
Approximately 30%
Proportion of weekly cannabis users who meet criteria for cannabis use disorder This rate is higher than for sporadic users and reflects the increased risk with frequent use.